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rabbit anti mgl  (Developmental Studies Hybridoma Bank)


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    Structured Review

    Developmental Studies Hybridoma Bank rabbit anti mgl
    Rabbit Anti Mgl, supplied by Developmental Studies Hybridoma Bank, used in various techniques. Bioz Stars score: 93/100, based on 7 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/rabbit+anti+mgl/10__1074_slash_jbc__m115__671842-70-35-45?v=Developmental+Studies+Hybridoma+Bank
    Average 93 stars, based on 7 article reviews
    rabbit anti mgl - by Bioz Stars, 2026-07
    93/100 stars

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    Fabp1 gene ablation (LKO) confers on high fat diet (HFD) the ability to decrease brain <t>2-monoacylglycerol</t> levels. All conditions were as in Figure 1 except that LC-MS analysis of 2-monoacylglyerols was performed using deuterated internal standards (Cayman Chemical) as described in Materials and Methods to quantify (A) 2-arachidonoylglycerol (2-AG), (B) 2-oleoylglycerol (2-OG), and (C) 2-palmitoylglycerol (2-PG). Results are expressed as the molar ratio of each NAE in High-Fat/Control diet and presented as mean ± SEM (n = 8); *, P < 0.05 for LKO vs WT; #, P < 0.05 for female (F) vs male (M).
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    Fabp1 gene ablation (LKO) confers on high fat diet (HFD) the ability to decrease brain 2-monoacylglycerol levels. All conditions were as in Figure 1 except that LC-MS analysis of 2-monoacylglyerols was performed using deuterated internal standards (Cayman Chemical) as described in Materials and Methods to quantify (A) 2-arachidonoylglycerol (2-AG), (B) 2-oleoylglycerol (2-OG), and (C) 2-palmitoylglycerol (2-PG). Results are expressed as the molar ratio of each NAE in High-Fat/Control diet and presented as mean ± SEM (n = 8); *, P < 0.05 for LKO vs WT; #, P < 0.05 for female (F) vs male (M).

    Journal: Journal of neurochemistry

    Article Title: Fabp1 GENE ABLATION INHIBITS HIGH FAT DIET-INDUCED INCREASE IN BRAIN ENDOCANNABINOIDS

    doi: 10.1111/jnc.13890

    Figure Lengend Snippet: Fabp1 gene ablation (LKO) confers on high fat diet (HFD) the ability to decrease brain 2-monoacylglycerol levels. All conditions were as in Figure 1 except that LC-MS analysis of 2-monoacylglyerols was performed using deuterated internal standards (Cayman Chemical) as described in Materials and Methods to quantify (A) 2-arachidonoylglycerol (2-AG), (B) 2-oleoylglycerol (2-OG), and (C) 2-palmitoylglycerol (2-PG). Results are expressed as the molar ratio of each NAE in High-Fat/Control diet and presented as mean ± SEM (n = 8); *, P < 0.05 for LKO vs WT; #, P < 0.05 for female (F) vs male (M).

    Article Snippet: Anti-rabbit fatty acid transport protein 1 (FATP-1; sc-25541), monoclonal anti-mouse fatty acid binding protein-3 (FABP3; sc-58275), as well as rabbit polyclonal anti-rabbit fatty acid binding protein-7 (FABP7; sc-30088), monoclonal anti-mouse N-acylethanolamide hydrolyzing acid amidase (NAAA; sc-100470), monoclonal anti-mouse β-Actin (sc-47778), rabbit polyclonal anti-monoacylglycerol lipase (MGL, sc-134789) and anti- diacylglycerol lipase α (DAGLα; sc-133307) were from Santa Cruz Biotech (Santa Cruz, CA).

    Techniques: Liquid Chromatography with Mass Spectroscopy

    Fabp1 gene ablation (LKO) differentially impacts the ability of high fat diet (HFD) to alter serum N-acylethanolamide (NAE) and 2-monoacylglycerol (2-MG) levels. All conditions were as in Figures 1 and ​and22 except that NAE and 2-MG were determined in serum by LC/MS as described in Materials and Methods. Panels refer to: (A) arachidonoylethanolamide (AEA), (B) oleoylethanolamide (OEA), (C) palmitoylethanolamide (PEA), (D) docosahexaenoylethanolamide (DHEA), (E) 2-arachidonoylglycerol (2-AG), (F) 2-oleoylglycerol (2-OG), and (G) 2-palmitoylglycerol (2-PG). Results are expressed as the molar ratio of each NAE or 2-MG in High-Fat/Control diet and presented as mean ± SEM (n = 8); *, P < 0.05 for LKO vs WT; #, P < 0.05 for female (F) vs male (M).

    Journal: Journal of neurochemistry

    Article Title: Fabp1 GENE ABLATION INHIBITS HIGH FAT DIET-INDUCED INCREASE IN BRAIN ENDOCANNABINOIDS

    doi: 10.1111/jnc.13890

    Figure Lengend Snippet: Fabp1 gene ablation (LKO) differentially impacts the ability of high fat diet (HFD) to alter serum N-acylethanolamide (NAE) and 2-monoacylglycerol (2-MG) levels. All conditions were as in Figures 1 and ​and22 except that NAE and 2-MG were determined in serum by LC/MS as described in Materials and Methods. Panels refer to: (A) arachidonoylethanolamide (AEA), (B) oleoylethanolamide (OEA), (C) palmitoylethanolamide (PEA), (D) docosahexaenoylethanolamide (DHEA), (E) 2-arachidonoylglycerol (2-AG), (F) 2-oleoylglycerol (2-OG), and (G) 2-palmitoylglycerol (2-PG). Results are expressed as the molar ratio of each NAE or 2-MG in High-Fat/Control diet and presented as mean ± SEM (n = 8); *, P < 0.05 for LKO vs WT; #, P < 0.05 for female (F) vs male (M).

    Article Snippet: Anti-rabbit fatty acid transport protein 1 (FATP-1; sc-25541), monoclonal anti-mouse fatty acid binding protein-3 (FABP3; sc-58275), as well as rabbit polyclonal anti-rabbit fatty acid binding protein-7 (FABP7; sc-30088), monoclonal anti-mouse N-acylethanolamide hydrolyzing acid amidase (NAAA; sc-100470), monoclonal anti-mouse β-Actin (sc-47778), rabbit polyclonal anti-monoacylglycerol lipase (MGL, sc-134789) and anti- diacylglycerol lipase α (DAGLα; sc-133307) were from Santa Cruz Biotech (Santa Cruz, CA).

    Techniques: Liquid Chromatography with Mass Spectroscopy